A Change Of Heart: What Your Cardiologist Didn't Mention

Clogged arteries from fatty deposits (including cholesterol), begin as an accumulation of positively-charged heavy metals such as mercury and lead in smooth muscle cells beneath the endothelium, (the inner vessel wall). Blood vessel walls are supposed to be negatively-charged when all's well. Negative charge causes negative blood cells to flow by without sticking to the wall. More accurately, they're propelled by repulsive, vortical, and expansionary forces.

But, when heavy metals (which are positively charged), build up in cells beneath the interior vessel wall, the vessel wall changes polarity. It changes from negative to positive charge locally, which is the polarity of injury, and the signal that calls healing mechanisms to the area. If you pinch yourself, the release of histamine immediately turns the area positive.

Positive charge on the vessel wall then causes (negative) platelets to stick to it. If the injury were a cut, platelets plug the vessel to stop the bleeding. Platelets then tell fibrocytes to attach themselves to the positive endothelium to make a mesh. We recognize the mesh as a scab.

Cholesterol then impregnates the mesh to form a soft plaque. If enough bacteria find food and lodging in the plaque, the body tries to wall it off with calcium to minimize the damage done to other tissues. This causes the plaque to solidify into a hard plaque.